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Enterobactin inhibits microbiota-dependent activation of AhR to promote bacterial sepsis in mice
, SURMOUNT-1 17,8 (19,3 16,3) 15 (n = 630) (n = 643) [6]
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An analysis of the GLP-1 receptor revealed not only the aforementioned molecular details related to the interactions, but also the prerequisite amino acid residue that explained the species selectivity in binding of non-peptide molecules [40,41]
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