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To address this question, we mutated Tyr110 in the CPYS-motif of ScGrx7 as a candidate residue that might contribute to the glutathione activator site (see Discussion for details) and performed a preliminary study with wild-type ScGrx7 as a control
Additionally, statins and liver X receptor antagonists suppress cholesterol biosynthesis and reduce mevalonate availability, thereby preventing mTOR-mediated trained immunity (96, 187)
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The enzyme cholinephosphotransferase catalyzes this reaction, leading to the formation of phosphatidylcholine and the release of CMP (cytidine monophosphate)
