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Genetic disruption of the multifunctional CD98/LAT1 complex demonstrates the key role of essential amino acid transport in the control of mTORC1 and tumor growth

Abstract Background: Although oxidative stress is known to contribute to cancer, and endogenous and exogenous antioxidants are thought to prevent tumorigenesis by suppressing oxidative stress-induced DNA damage, antioxidants have also been reported to show negative effects on tumor formation, necessitating characterization of the causal associations between antioxidants and cancer risk

In fact, there are other medications that tackle the same sorts of issues, potentially even in a synergistic way

Firstly, GA significantly upregulates key drug-metabolizing enzyme genes (e.g., Cyp2b13 , Cyp2c69 , Cyp3a44 , Cyp3a16 , and Fmo3 ) that are downregulated by TGT, thereby enhancing liver detoxification capacity (Xie et al., 2016)