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Metabolites, through post-translational modifications of metabolite-sensitive protein (i.e
But these side effects are very rare
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GLP1/E therapy led to upregulation of pathways involved in autophagy and vesicle-mediated transport, suggesting that this intervention enhances hypothalamic autophagy, a mechanism that may contribute to the metabolic improvement observed in GLP1/E-treated animals
The glp-1(q35) has a premature stop codon, which results in a truncated GLP-1/Notch protein that lacks a negative regulatory domain (ProGluSerThr [PEST]) (Fig
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