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The mechanisms are biologically plausible
In gastrointestinal models, researchers observed that BPC-157 is associated with increased expression of tight junction proteins and mucosal protective factors
The parent compound Epithalon (AEDG) lacks the protective end-modifications
In vitro studies demonstrate that BPC-157 promotes survival of cells exposed to various stressors including oxidative damage, toxic substances, and serum deprivation
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