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NF-B Activation and Chronic Inflammation GSH normally maintains NF-B in its inactive state by preserving the redox status of IB kinase within immune cells
In addition, compared with nasal mucosa, the oral epithelial barrier presents relatively lower overall permeability
Current understanding attributes ferroptosis in ischemic stroke to several factors, including intracellular iron accumulation, lipoxygenase activation, GSH depletion, GPX4 inactivation, and system Xc disruption (130) ( Figure 3 )
But they work in very different ways, and choosing between them is not always straightforward
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