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It is not clear whether the replacement of melanocytes of the basal cell layer of the oral epithelium lost to physiological processes of apoptosis or senescence, or to mechanical, thermal, or chemical injury is the result of cell division of mature melanocytes in the basal cell layer of the oral epithelium which still retain their capacity of replication, or a result of migration from their local niche reservoirs of melanocyte stem/progenitor cells and their subsequent proliferation [6]
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The results indicated that TMAO could increase the intracellular Ca 2+ level, up-regulate the expression of ANP and MYH7, and down-regulate SERCA2a expression, which could be reversed by overexpressing of SERCA2a and BAPTA-AM
By serving as a selective agonist for amylin and calcitonin receptors, it enables researchers to dissect the downstream molecular events associated with satiety, insulin sensitivity, and nutrient partitioning
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