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Therefore, it is now understood that the delayed or impaired healing of wounds that occur in patients with diabetes is caused by both molecular and cellular abnormalities involving a reduction in the hosts immune response, neuropathy, damage from reactive oxygen species, and advanced glycation end products, abnormalities in fibroblast and epidermal cell functions, hypoxia, impaired angiogenesis, and elevated levels of MMPs

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PubMed 16352683) remains the definitive human pharmacology study: CJC-1295 (with DAC) in 57 healthy adults produced 210 GH elevation and 1.53 IGF-1 elevation sustained for 911 days

10.1016/j.biochi.2014.01.010 199 StipanukM