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Instead, it is designed solely to activate the innate repair receptor (IRR) , the bodys primary switch for tissue protection and anti-inflammation.According to research published in Molecular Pain , ARA-290 is disease-modifying. It works by quieting microglia , the immune cells in the nervous system that drive chronic pain when overactive

The result is preserved intracellular pools of nicotinamide ( NAD+ salvage substrate) and SAM ( methyl-donor pool)

Small intestine conditions such as celiac disease, Crohns disease, parasite or bacterial growth

restoring these levels helps offset that decline and supports a balanced, regenerative state