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Recent studies have demonstrated that BMSC-Exos can promote neurological recovery in ASCI by inhibiting ferroptosis through the Nrf2/GTP cyclohydrolase I (GCH1)/5,6,7,8-tetrahydrobiopterin (BH4) signaling axis, reducing oxidative stress, and enhancing the expression of ferroptosis suppressors [146]
Additionally, analogs of the peptide have been shown to decrease infarct size in ischemia-reperfusion injuries and strengthen antioxidant defenses, highlighting their potential to protect brain health and support recovery from neurological damage (7)
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