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Induction of thermogenic adipocytes: molecular targets and thermogenic small molecules
Rising diabetes and obesity prevalence, strong clinical outcomes, patient awareness, insurance coverage, and demand for effective, convenient metabolic therapies drive growth
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Erad interplay with ER-phagy Along with ERAD-mediated clearance of misfolded, unfolded, or incompletely assembled proteins, there is increasing recognition that, via lysosomal degradation, selective autophagy of the ER (ER-phagy) also serves to dispose of misfolded proteins or protein aggregates unrecognizable by the ERAD or too large to undergo translocation from the ER into the cytoplasm for ERAD
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