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A specific oxidoreductase, previously known as apoptosis-inducing-factor mitochondrial-2 (AIFM2), capable of recycling reduced ubiquinol (Co-enzymeQ 10 H 2 ) from ubiquinone at the expense of NAD(P)H, has been presented as a potential ferroptosis inhibitor due to the fact that its overexpression complements the loss of GPx4 in PFA1 and human fibrosarcoma (Figure 2)
Nevertheless, this work validates the potential of topical antioxidant delivery as a therapeutic strategy for RP
14,15,16 Understanding its multifaceted functions and mechanisms is crucial for deciphering its implications in health and disease
PMC9915969 review of GI adverse events on dual GIP/GLP-1 receptor agonists
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